Soft Tissue Injuries of the Hand

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Practice Essentials

The pathophysiology of soft tissue injuries of the hand is diverse. The most common mechanisms of injury are blunt trauma (eg, crush injury, contusions, abrasions), laceration, avulsion, ring avulsion, and burns. Besides skin and superficial tissues, the many muscles, ligaments, and tendons of the hand are vulnerable to injury, as are the nerves and blood vessels that supply these structures. Damage to these structures may create permanent functional and/or sensory deficits specific to the site of injury.[1, 2, 3]

Hand injuries are common and account for 5-10% of emergency department (ED) visits nationwide. In one study of upper extremity injuries and infections treated in EDs in the United States, the majority (37%) were soft tissue contusions. Over 50% of patients were male, and the most common age group was 18-44 years (44%), followed by those younger than 17 years (24%) and those 45-64 years (21%).[4]  Trauma accounts for the majority of these injuries. However, patients also present with complaints that are secondary to infection, burns, or overuse.

The complexity of the hand and the similarities in clinical presentation of different injuries make understanding of hand anatomy and function, good physical examination skills, and knowledge of indications for treatment indispensable for the emergency physician.

Complications of soft tissue hand injuries may include pain, stiffness, abscess, decreased range of motion, scar formation, and, rarely, loss of a digit or hand.

Terminology

Thorough knowledge of the anatomy and functions of the hand is required for proper diagnosis and treatment. Use of proper terminology prevents confusion that may compromise the care of patients with hand injuries. The following is a brief review of standard terminology and key anatomic structures:

Standard terminology also applies to motions and positions of the hand and digits, such as the following:

Anatomy

Three creases are present on the palmar surface of the digits. The distal and middle palmar creases correspond to the distal interphalangeal (DIP) and proximal interphalangeal (PIP) joints, respectively. The proximal digital palmar crease does not overlie a joint, as the MCP is more proximal. The long thenar crease partially encircles the thenar eminence and overlies the metacarpophalangeal (MCP) joint.

The wrist is composed of 8 carpal bones arranged in 2 rows of 4. The flexor retinaculum together with the carpal bones forms the carpal tunnel. The median nerve passes through the carpal tunnel with the tendons of the flexor digitorum profundus and superficialis. The ulnar nerve enters the hand passing between the hook of the hamate bone and the pisiform bone in the Guyon canal.

The metacarpal bones articulate with the wrist at the carpometacarpal (CMC) joints. The MCP joints are formed by the articulation of the metacarpal bones with the proximal phalanges (see the image below). The heads of the metacarpals form the knuckles, which are seen dorsally with the closed fist. The thumb has only one interphalangeal (IP) joint, while the rest of the digits have PIP and DIP joints.



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Metacarpophalangeal joints of the digits

Each of the MCP, PIP, and DIP joints has collateral ligaments, which provide lateral stability, and a volar plate, which prevents hyperextension. The volar plate is damaged frequently in subluxation and dislocation injuries.

The blood supply to the hand is derived from the ulnar and radial arteries, which form the superficial and deep palmar arterial arches by anastomosis. In the absence of vascular disease, either artery alone is sufficient to perfuse the entire hand in most of the population.

The muscles of the hand are designated as intrinsic or extrinsic. Extrinsic muscle bellies are in the forearm, and their tendons insert into the hand, while intrinsic muscles both arise in and insert in the hand. The muscles of the hand and digits also are named according to their function as either flexors or extensors.

The forearm flexors are extrinsic muscles of the hand. These muscles arise from the medial epicondyle of the humerus and include the following:

The tendons of flexor carpi radialis (FCR), palmaris longus, and flexor carpi ulnaris (FCU) are visible in the forearm (see the image below). The palmaris longus is absent in about 14% of the population. The median nerve lies between the palmaris longus and the flexor carpi radialis (to the ulnar side of the FCR). The flexor carpi ulnaris is a good landmark to locate the ulnar nerve and artery, which lie to the radial side of the FCU.



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Volar tendons at the wrist. These can be used as landmarks for injections.

Flexion of the fingers is controlled by the flexor digitorum profundus and superficialis muscles. Both of the finger flexors lie on the ulnar side of the wrist with the median and ulnar nerves and the ulnar artery. The flexor carpi ulnaris and radialis flex the wrist when acting together and cause deviation to their respective active sides when contracting separately.

Branches of the median and ulnar nerves innervate all the intrinsic muscles of the hand. They can be divided into 3 groups: thenar (thumb), hypothenar (little finger), and lumbricals.

The thenar eminence is formed by the extensor pollicis brevis and the 3 short thenar muscles: the abductor pollicis brevis, flexor pollicis brevis, and opponens pollicis. These muscles have short tendons that insert onto the proximal phalanx of the thumb. They are innervated by the recurrent branch of the median nerve. The superficial location of this branch renders it vulnerable to seemingly trivial trauma to the thenar eminence.

The forearm extensors consist of 11 muscles that extend the wrist, hand, and digits (see the image below). The forearm extensors pass into the hand in 6 compartments. All forearm extensors arise from the lateral epicondyle of the humerus. They are innervated by the radial nerve.



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Sagittal section of extensor compartments

The median, ulnar, and radial nerves supply all of the sensory and motor innervation to the hand. The superficial volar and dorsal distributions of the sensory nerves are shown in the images below. The median nerve enters the hand via the carpal tunnel and often is involved in carpal tunnel syndrome. The median nerve sends motor fibers to the 3 short thenar muscles and the first and second lumbricals. The ulnar nerve sends motor fibers to the hypothenar muscles, the ulnar 2 lumbricals, the adductor pollicis, and all of the interosseous muscles. The radial nerve sends no motor branches to the intrinsic muscles of the hand.



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Superficial volar sensation of the hand



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Superficial dorsal sensation of the hand

Pathophysiology

Nerve injuries

Blunt, penetrating, and crush injuries to the hand result in nerve damage.[5] Nerve injury is divided into 3 types, as follows:

Dislocations

The DIP joint is stabilized not only by collateral ligaments but by adjacent flexor and extensor tendons, making dislocations of this joint uncommon. If dislocation does occur, it usually is directed dorsally and often is associated with an open wound. DIP joint dislocations are detected easily by physical examination.

The ligaments of the PIP joints are the most commonly injured in the hand. Dorsal dislocations are the most common and usually are the result of a blow to the extended digit, causing a combination of axial loading and dorsal deviation. Volar dislocations are uncommon because the joint does not resist motion in this direction. Lateral dislocation is the result of a tangential load applied to the extended digit that ruptures a collateral ligament and disrupts the volar plate. Ulnar deviation, with rupture of the radial collateral ligament, is more common than radial deviation.

Dislocation of the MCP joint is uncommon, but when it occurs, deviation is usually dorsal. The common mechanism of injury is the application of a dorsally directed force that is sufficient to rupture the volar plate. Dorsal dislocations in 60-90° of hyperextension and without intervening soft tissue are simple dislocations. Complex dislocations have the volar plate entrapped between the metacarpal and the proximal phalanx. Complex dislocations are less striking in their clinical presentation but are more serious injuries.

The IP joint of the thumb is very stable and seldom injured. Dislocations usually are dorsal and often open. The MCP joint of the thumb is one of the most frequently injured joints. Injury is most commonly caused by a hyperextension force sufficient to rupture the volar plate and cause dorsal dislocation. As in MCP joints of the other digits, dorsal dislocation of the MCP joint may be a simple subluxation or complex dislocation. The complex dislocation is complicated by entrapment of the proximal phalanx.

Ligament injuries/sprains

Joints of the digits are stabilized by the combination of collateral ligaments and the volar plate. Stretching or partial tearing of the ligaments results in a sprain. The volar plate may be injured alone or in combination with the collateral ligaments. The common mechanism for an isolated volar plate injury is hyperextension during an axial load. Pain location is a good indicator of the site of injury. For example, lateral pain suggests collateral ligament injury, whereas pain on the palmar surface of the joint suggests volar plate injury. Loss of stability is more commonly associated with joint dislocation.

Sprains of the PIP and MCP joints produce pain and swelling but may lead to little or no instability. They are classified as first, second, or third degree. If the joint does not open at all but has pain with stressing of a ligament, the injury is first degree. A joint that is opened slightly in the ulnar or radial direction is defined as having a second-degree injury. This finding suggests a unilateral collateral ligament tear. A joint that is opened by at least 3-5 mm must have damage to at least 2 of the 3 structures stabilizing the joint (ie, volar plate, 2 collateral ligaments). This is referred to as a third-degree sprain or an unstable joint. Serious ligamentous injuries occur frequently and often are misdiagnosed because a mild sprain may have a similar presentation. Sequelae from missed ligamentous injuries range from chronically painful to chronically unstable or deformed joints.

Sprains of the MCP joint are rare because of the anatomy of the joint, the laxity of the collateral ligaments, and the protection afforded the joints by surrounding structures. Hyperextension of the extended digit is the most common mechanism causing sprains. Diagnosis is indicated by a stable but painful edematous joint.

The ability to hold objects between the thumb and 4 fingers is an essential function of the hand and depends upon an intact ulnar collateral ligament (UCL). Injury to the UCL is known as the gamekeeper's thumb or skier's thumb. This is because Scottish gamekeepers frequently damaged their UCLs killing game. The head of a small animal was placed between the thumb and index finger, and a hyperextension/longitudinal traction force was applied to the animal's cervical spinal cord by abruptly yanking the lower extremities. In a certain percentage of these procedures, the UCL of the gamekeeper was disrupted.

In modern times, skiing is the activity that most often causes UCL injury. However, a history of a missed punch, a fall onto the thumb, or the forceful removal of an object from the flexed hand also should be considered suggestive of UCL injury.

The common mechanism of injury is the forceful abduction of the thumb. Any patient with pain in the distribution of the UCL or inability to forcefully oppose the thumb has an injury of the UCL until proven otherwise.

Rupture of the radial collateral ligament of the thumb is much less common than UCL rupture. The mechanism of injury is forceful adduction of the thumb in any position.

Tendon injuries

The extensor tendons' superficial location predisposes them to injury from seemingly trivial lacerations as well as avulsions, crushes, and burns.[2, 3, 6, 7] Flexor tendon injuries can be caused by lacerations. Tendon injuries also may be sustained as the result of forced hyperextension or forced flexion of an extended digit. Injuries may include complete or partial transection, avulsion, or maceration. Whenever a tendon is damaged, particularly with an open injury, the vessels and nerves that are in close proximity may be injured.[8, 9]

Understanding that a tendon may be 70-90% lacerated and still functional is critical. Damage to these tendons may result in such findings as boutonnière deformity and mallet finger (see the images below).[10, 6]



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Boutonnière deformity due to closed central tendon rupture



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Boutonnière deformity



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Mallet finger due to loss of central extensor tendon to the distal phalanx

History

The general history for soft tissue hand injuries includes age, hand dominance, occupation/hobbies, history of previous hand problems, other past medical history (especially diabetes, vascular problems), and smoking history.

In cases involving trauma, ascertain when and where the injury occurred to determine the likelihood of severe injury and probability of contamination with foreign matter.

How the trauma was sustained provides clues to the most likely injury. For example, a water skier who injured a hand when the towing line was removed forcefully from that hand is likely to have an injury to the flexor tendon mechanism.

Determine the posture of the hand at the time of the injury. Structures in the hand slide with movement. The tissue under a bruise or laceration may not be the same tissue that was present when the injury was sustained because of movement of structures in the hand (eg, extensor tendons injured with the digits in flexion may not be visible in the wound when digits are extended).

Past history of treatment or surgery in the hand should be noted.

Physical

The entire upper extremity should be exposed. Note any of the following findings:

Look for differences in flexion/extension in the relaxed hand. The relaxed hand is in moderate flexion. The digits on both sides should be in about the same amount of flexion. The little finger usually is in more flexion than the other fingers. If a digit has a marked difference in flexion, the examiner should be suspicious for tendon injury. This finding may be useful in identifying the injury of the patient who is a poor historian.

Dry patches of skin may indicate loss of innervation.

Dimpling over the thenar eminence suggests complex dislocation of the MCP joint of the thumb.

Check range of motion in every joint in the hand, shoulder, and elbow. Ability to pronate and supinate the forearm should be tested actively and passively.

Test grip and pinch strength.

The best way to diagnose a tendon injury in an open wound is by direct visualization during a thorough exploration of the wound.

Examination of extrinsic flexors

Each of these tests is performed with and without resistance.

Flexor pollicis longus: Instruct the patient to bend the tip of the thumb against resistance.

Flexor digitorum profundus: While holding the PIP joint in extension, instruct the patient to bend the tip of the finger.

Flexor digitorum superficialis: While stabilizing the rest of the fingers to block the action of flexor digitorum profundus, instruct the patient to bend the middle joint of the finger.

Palpate the tendons of flexor carpi ulnaris, flexor carpi radialis, and palmaris longus (which is not present in about 15% of individuals) while the patient holds the wrist and fingers in hyperflexion.

Extrinsic extensors

Extrinsic extensors arise from the forearm and insert into the hand. The extrinsic extensors pass from the wrist to the hand in 6 tendon compartments, as follows:

Extrinsic extensors may form adhesions secondary to old trauma. This phenomenon is referred to as extensor tightness. Extensor tightness is evaluated by passive extension of the MCP joint and flexion of the PIP joint with the wrist in anatomic position. The PIP joint should flex. Repeat the test with the MCP joint in passive flexion. If the PIP joint will flex when the MCP joint is extended but not when it is flexed, adhesions are present in the extensors, stopping the simultaneous flexion of the finger MCP and PIP joints.

Intrinsic muscles

Intrinsic muscles have their origins and insertions in the hand. They include thenar muscles, lumbricals, interosseous muscles, and hypothenar muscles.

The thenar muscles include the abductor pollicis brevis, opponens pollicis, and flexor pollicis brevis. These muscles oppose the thumb. Test by asking the patient to place the back of the hand on the table and raise the thumb until it rests perpendicular to the hand or to touch the thumb to each finger. Palpate the muscles of the thumb and compare findings to the other side.

For the adductor pollicis, test separately by asking the patient to grasp 2 ends of a piece of cloth and hold them tightly between the thumb and index finger. Flexion of the thumb at the IP joint is called the Froment sign and indicates damage to the adductor pollicis or the ulnar nerve.

Interosseous and lumbrical muscles flex the MCP joints and extend the IP joints. The interosseous muscles are innervated by the ulnar nerve. Evaluate by asking the patient to spread the fingers and by checking the resistance to ulnar and lateral deviation of each digit in abduction. The extrinsic extensors can be used to abduct-adduct the digits if the interossei are deranged. To block their action, ask the patient to place the palm on the table and to hyperextend the digits at the MCP joints.

The hypothenar muscles include the abductor digiti minimi, flexor digiti minimi, and opponens digiti minimi. Evaluate by asking the patient to deviate the small fingers in an ulnar direction. Palpate the hypothenar eminence while the digit is in abduction.

Joint assessment

The stability of a joint is assessed by active and passive motion. Pain causes some patients to consciously or unconsciously limit the range of motion of an injured joint. Therefore, administering a digital block may be necessary prior to assessing joint stability.

Evaluate stability by applying anterior, posterior, radial, and ulnar stress to each IP and MCP joint in the extended and flexed positions. Evaluation in the flexed and extended positions is necessary, as the volar plate may stabilize a dislocated or subluxated joint in certain positions.

Sensory examination

Thoroughly inspect the skin. Denervated areas are often dry because of loss of sympathetic innervation. This may be useful in children or other patients who cannot give a history.

The immersion test also may be useful in patients who cannot give a history. Denervated skin does not wrinkle after being exposed to water for 5-10 minutes.

Two-point discrimination is the best overall test of sensory function. The distance between 2 prongs, beginning at 6 mm, is increased and decreased during the course of the exam. Abnormal discrimination values are less than 6 mm static and less than 3 mm moving. An abnormal discrimination examination implies axonal loss and sensory deficit that may be due to laceration, compression, or contusion of the nerve. If exam findings are abnormal, repeat the test on the unaffected side because the sensitivity and specificity vary from patient to patient.

Circulation

Look for color changes in the nails and skin of the hand.

The Allen test has variable sensitivity, but it may be used to help assess perfusion to the hand. Compress radial and ulnar arteries at the wrist. Instruct the patient to open and close the fist to exsanguinate the hand. Have the patient open the hand. Release the radial artery. If the hand fills with blood within 5 seconds, the radial artery is patent. Repeat the test for the ulnar artery.

Imaging Studies

Plain-film radiographs of the hand or wrist should be obtained when a patient presents with a soft tissue injury suggestive of fracture or an occult foreign body. Of particular concern are lacerations from glass. Radiography is indicated in dislocation of a DIP joint to rule out fracture. In dislocation of an MCP joint, the posterior-anterior view shows marked widening of the joint. Sesamoid bones seen inside the joint space on radiographs are diagnostic of complex MCP joint dislocation.

MRI has been shown to be highly sensitive in detecting ruptured tendons. However, it does not have a role in emergent management of hand wounds.

Ultrasonography has a growing role in locating foreign bodies and in evaluating soft tissues (see the image below). It can be used to detect ruptured tendons and ulnar collateral ligament injuries to the thumb. Also, the dynamic function of tendons can be assessed noninvasively with ultrasonography. Ultrasound evaluation in the long axis with use of dynamic imaging allows visualization of the ulnar collateral ligament and adductor pollicis aponeurosis. Ultrasound has also been found to be accurate in diagnosing Stener lesions.[11, 12]

One prospective study found that high-resolution ultrasound (5-13 Mhz) is often superior to clinical diagnosis of many disorders, including ganglion cysts, tendinopathies, and retained foreign bodies.[13] However, it is less accurate with diagnosis of solid lesions.[13, 14]



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This image shows an ultrasound of the flexor tendon of a finger with the structures labeled. It was performed using a water bath with the water as the....

 

Emergency Department Care

The prevalence and acute nature of soft tissue injuries to the hand require that the emergency physician understand the principles of evaluation and treatment. Mutilation of the hand may be taken to include injuries that result in significant damage to several tissues at once. The patterns of these injuries are highly variable because of the numerous permutations and combinations of the severity, extent, and the number of tissues involved that the treatment.[15]  The final functional outcome of each injury is unique and varies with the severity of structural damage and contamination, as well as surgery and rehabilitative care. The overall goal of reconstruction is restoration of maximal function in the shortest possible time with the minimum number of procedures.[15]

Time considerations

The time that a patient was injured and the time of presentation to the ED should be recorded. Certain types of injuries require rapid response to prevent unfavorable outcomes. The following injuries require immediate treatment after diagnosis:

Lacerations

Skin wounds of the hand, although commonplace, should not be trivialized. They must be handled with a methodical and thorough approach to optimize outcome and minimize morbidity.

After the initial examination and a decision regarding need for radiologic studies, anesthetize the hand wound with buffered lidocaine. A digital block is preferable for digital injuries but only after a careful sensory exam, including 2-point discrimination.

Next, irrigate the wound profusely using clean or sterile water under pressure. Care should be taken not to infiltrate the wound with the irrigant.

Drape the wound and examine it carefully under proper lighting, including visualization of the entire wound during a full range of motion. Search vigorously for foreign bodies or evidence of tendon injuries.

To achieve hemostasis during wound exploration, fasten a sterile Penrose drain to the base of a digit. Do not use a rubber band, which can easily be overlooked and lead to an ischemic digit. Inflate a blood pressure cuff to over 200 mm Hg, then clamp the tube to achieve good hemostasis. Total tourniquet time in the ED should not exceed 2 hours.

Close the skin wound with a single layer of simple or horizontal mattress sutures.[7] Deep sutures should seldom, if ever, be placed in the ED because of the risk of infection and granuloma formation.

Hand wounds older than 6-8 hours should not be closed primarily, because of an increased likelihood of infections. Irrigate and explore such wounds and apply a sterile dressing. Recheck the wound in 2-4 days, with consideration of delayed primary closure at 4 days.

Similarly, most bite wounds and wounds sustained by blunt injury to another person's mouth (a "fight-bite") should not be closed primarily but should be given serial wound checks with delayed closure at 4 days if needed.

Antibiotic prophylaxis is indicated in human (including fight-bites) and cat bites and may be of benefit in dog bites as well. The use of antibiotics in other hand wounds is controversial but generally is best reserved for contaminated wounds and puncture wounds with possible retained foreign bodies.

Nerve injury

A completely disrupted nerve should be repaired microsurgically. However, in the acute setting, distinguishing the severity of nerve damage often is impossible. Consultation with a hand surgeon is advised.

Primary repair is optimal, but if delayed repair is advised because of a dirty wound, multiple injuries, or logistic constraints, the finger/hand involved should be splinted and the patient should receive prompt follow-up with a hand surgeon.

Sprains

Patients with an unstable joint need referral to a hand surgeon for nonurgent follow-up.

The collateral ligaments of the MCP joints are tensed when the joints are flexed and relaxed in extension, just the opposite of most other collateral ligaments. Prolonged immobilization in extension may shorten these ligaments and result in reduced mobility of the joints. Therefore, MCP joints usually are immobilized in 50-60° of flexion. PIP joints should be splinted in 20-30° of flexion.

Injuries to the collateral ligaments are treated according to the degree of injury:

Dislocations

For distal interphalangeal joints, radiographic studies are indicated to rule out fractures. Reduce the dislocation after administering a digital or metacarpal block. While holding the phalanx proximal to the injury, apply a distracting force along the longitudinal axis of the digit. While maintaining traction, hyperextend the phalanx (for dorsal dislocations) and bring it back to its normal anatomic position. Examine the joint thoroughly after reduction. Then immobilize the finger with an aluminum splint. If the joint is irreducible, consultation with a hand surgeon is required. Inability to reduce a digit may be the result of entrapment of the volar plate or an avulsion fracture in the joint space. Irrigation, debridement, bacterial prophylaxis, and wound closure are indicated for open wounds.[16]

For proximal interphalangeal joints, lateral and dorsal dislocations may be treated effectively with closed reduction. Anesthetize the digit by digital or metacarpal block. Metacarpal block may be the preferred technique because a digital block causes further swelling of the injured digit. While holding the phalanx proximal to the injury, apply a distracting force along the longitudinal axis of the digit. While maintaining traction, hyperextend the phalanx (for dorsal dislocations) and bring it back to its normal anatomic position. Inability to reduce a PIP dislocation may be the result of entrapment of the volar plate or an avulsion fracture in the joint space. Consultation with a hand surgeon is required. Thorough physical examination with active and passive range of motion is required after reduction. If the joint remains deviated by more than 20° compared to the unaffected side, surgical referral is indicated. If the joint is stable with active and passive range of motion, 3 weeks of immobilization followed by physical therapy is indicated. Pain and stiffness are likely sequelae and the patient should be forewarned. However, long-term prognosis is good.

For metacarpophalangeal joints, the recommended treatment of complex and volar dislocations is a gentle compression dressing and urgent consultation with a hand surgeon because they are likely to require open reduction. Reduction of simple dislocations of the MCP joint may be attempted by an emergency physician, although reduction often is unsuccessful. Entrapment of the metacarpal head between muscles and tendon on the palmar side of the hand often prevents closed reduction. After administration of a metacarpal or wrist block, flex the wrist to relax the flexor tendons. Flex the proximal phalanx while applying mild longitudinal traction. Use care to avoid hyperextension or excessive longitudinal force, which may open the joint space and allow entrapment of the volar plate. Following successful reduction, immobilize the hand in a planar splint and refer the patient to a hand surgeon.

For the interphalangeal joint of the thumb, evaluation and treatment of thumb IP joint injuries are similar to those for the IP joints of the fingers. After reduction, the joint should be immobilized in 20° of flexion for 3 weeks. For the metacarpophalangeal joint of the thumb, simple dislocations may be reduced following administration of a median nerve block. Flex and abduct the MCP joint and apply longitudinal force to the base of the proximal phalanx.  If this method is unsuccessful, flexion of the IP joint and wrist will relax the flexor pollicis longus tendon, which may be complicating the reduction. Thorough examination is necessary after reduction. If the joint is stable, immobilization of the MCP joint in 20° of flexion for 3 weeks is indicated.

Ligament injuries

For ulnar collateral ligament injuries of the thumb, whenever the history or clinical signs cause suspicion for UCL injury, stability of the collateral ligaments of the thumb must be assessed. Assessment usually requires a median nerve block. Compared to the undamaged side, if the thumb is deviated more than 20° or no firm endpoint to the joint opening can be appreciated, referral is prudent. If the thumb MCP joint is unstable or if a complete UCL tear is suspected, the thumb should be immobilized in a thumb spica splint and the patient should be referred to a hand specialist within a few days. If less than 20° of deviation is present as compared to the normal side and if a firm endpoint is appreciated, immobilization in a thumb spica splint should be undertaken.

Evaluation and treatment of radial collateral ligament injury is the same as for UCL injury.

Tendon injuries

The emergency physician should search carefully for tendon injuries. One British study showed significant deficiencies of emergency physicians in identifying tendon and nerve injuries with hand lacerations.[17]

The superficial location of extensor tendon injuries facilitates evaluation and permits repair in the ED. Partial tendon injuries (< 40-50% of the tendon width) usually do not require repair. They should be splinted and follow-up arranged with a hand surgeon.

Complete extensor tendon injuries can be repaired using 4.0 nonabsorbable suture material and a figure 8 or modified Kessler suture, with the knot buried on the palmar aspect of the tendon. However, this procedure does not need to be performed urgently, and closure of the skin, splinting of the hand, and referral to a hand surgeon for delayed repair is often the best option.

Flexor tendons are very sensitive to manipulation and prone to form adhesions. Restoration of a smooth gliding function is essential to future normal use of the hand. For this reason, primary repair should never be attempted in the ED. Repair should be done by a qualified hand surgeon in an operating room equipped for microsurgery.

Guidelines Summary

The American College of Radiology (ACR) guidelines for imaging acute hand and wrist trauma includes the following key recommendations[18] :

Author

Andrea B Lese, MD, Assistant Professor, Department of Orthopedics, West Virginia University School of Medicine

Disclosure: Nothing to disclose.

Coauthor(s)

Kenneth R Chuang, MD, Attending Physician, Emergency Department, Long Beach Memorial Medical Center and Miller Children's Hospital

Disclosure: Nothing to disclose.

Wyatt Decker, MD, Vice President and Chief Executive Officer, Mayo Clinic Campus, Arizona

Disclosure: Nothing to disclose.

Specialty Editors

Francisco Talavera, PharmD, PhD, Adjunct Assistant Professor, University of Nebraska Medical Center College of Pharmacy; Editor-in-Chief, Medscape Drug Reference

Disclosure: Received salary from Medscape for employment. for: Medscape.

Eric L Legome, MD, Professor and Chair, Department of Emergency Medicine, Mount Sinai St Lukes and Mount Sinai West; Vice Chair of Academic Affairs, Department of Emergency Medicine, Icahn School of Medicine at Mount Sinai

Disclosure: Nothing to disclose.

Chief Editor

Trevor John Mills, MD, MPH, Chief of Emergency Medicine, Veterans Affairs Northern California Health Care System; Professor of Emergency Medicine, Department of Emergency Medicine, University of California, Davis, School of Medicine

Disclosure: Nothing to disclose.

Additional Contributors

Dan Danzl, MD, Chair, Professor, Department of Emergency Medicine, University of Louisville Hospital

Disclosure: Nothing to disclose.

Acknowledgements

The authors and editors of Medscape Reference gratefully acknowledge the contributions of previous author, Kahill Glenn, MD, to the development and writing of this article.

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Metacarpophalangeal joints of the digits

Volar tendons at the wrist. These can be used as landmarks for injections.

Sagittal section of extensor compartments

Superficial volar sensation of the hand

Superficial dorsal sensation of the hand

Boutonnière deformity due to closed central tendon rupture

Boutonnière deformity

Mallet finger due to loss of central extensor tendon to the distal phalanx

This image shows an ultrasound of the flexor tendon of a finger with the structures labeled. It was performed using a water bath with the water as the conduction agent. (Image courtesy of Dr. Christopher Moore and Dr. Michael Osborne.)

Metacarpophalangeal joints of the digits

Volar tendons at the wrist. These can be used as landmarks for injections.

Sagittal section of extensor compartments

Superficial volar sensation of the hand

Superficial dorsal sensation of the hand

Boutonnière deformity due to closed central tendon rupture

Boutonnière deformity

Mallet finger due to loss of central extensor tendon to the distal phalanx

This image shows an ultrasound of the flexor tendon of a finger with the structures labeled. It was performed using a water bath with the water as the conduction agent. (Image courtesy of Dr. Christopher Moore and Dr. Michael Osborne.)